By L. Kayor. Norfolk State University. 2018.
Pitman decided it was ﬁnally time to take matters into his own hands discount 20 mg levitra soft otc erectile dysfunction exercises wiki, even though urology was not his ﬁeld of expertise buy 20 mg levitra soft fast delivery impotence grounds for divorce states. He engaged in some research at the hospital library as well as on the Internet. This led him to consider that he might have a hormonal problem, so he sought the help of an endocrinologist. Like the urologists before him, the endocrinologist had never heard of this particular mystery malady. However, he had heard about our Eight Steps to Self-Diagnosis and advised Dr. Painful Erections • Quality and Character: These erections are very hard and unrelenting. For my ﬁftieth birthday, I bought myself a Harley-Davidson and took a trip across several states, much to my wife’s dismay. Except for my cholesterol and triglycerides, which were high, I was in very good health. The doctor told me to lose some weight and to watch my sweets and alcohol intake. He also prescribed cholesterol medication and Rogaine for male pattern baldness. On the other hand, noth- ing seems to make it worse, but it doesn’t stop. Step Four: Do a Family Medical History and Determine If You Have or Had Any Blood Relatives with a Similar Problem. Pitman did what amounted to a medical genealogy chart, which was quite lengthy. This does not appear to be genetic, however, because my cousin’s condition was the result of an inﬂammation of the urethra. My urologist had already eliminated this as the cause of my problem along with other possible causes, scuch as a reaction to medication used to counteract impo- tence such as Viagra. Step Five: Search for Other Past or Present Mental or Physical Problems. Other than my painful erection problem, I had a cervical sprain after an automobile accident in my twenties, kidney stones several years ago, and now a high cholesterol problem which is currently being treated with med- ication. Could my new lifestyle/hobby—motorcycling—have anything to do with my medical mys- tery? Also, in trying to keep an open mind and thinking about my belief systems, there is a slight possibility that the symptoms do not indicate a dis- ease or condition, which is why I can’t ﬁnd a diagnosis; maybe they are a side effect of some medication. I have listened to my patients who have from time to time complained about side effects of medications I prescribed, and I dismissed them if I couldn’t ﬁnd any literature or ﬁndings that would sup- port their complaints. Now, since I cannot ﬁnd a diagnosis for my condi- tion—it’s not in the medical books—I am wondering if I have been too quick to dismiss this issue. Maybe I’d better investigate this further even though I have not seen my condition listed as a side effect. It may not be an independent disease or condition, and my symptoms have to be caused by something! Step Eight: Take Your Notebook to Your Physician and Get a Complete Physical Exam. Pitman a physical exam- ination because they were located in different states, but the endocrinolo- gist and urologists had already done so. Pitman raised two excellent questions that were brought forward as a result of doing the Eight Steps: whether motorcycling could be causing some trauma and possibly be a precipitating cause of his problem, and whether his problem was iatro- genic (medically induced by a medication).
The 37 THE REGULATION OF LIFESTYLE proportion of doctors who smoked fell from nearly 40 per cent in 1951 to just above 20 per cent ten years later (RCP 1962:11) trusted 20mg levitra soft erectile dysfunction pills at cvs. In the second phase 20mg levitra soft with mastercard erectile dysfunction protocol book, during the 1960s and 1970s, the campaign against smoking went public. This phase opened with the publication in Britain of the Royal College of Physicians’ pamphlet Smoking and Health, and in the USA, with the Surgeon General’s Report of the same title (RCP 1962; US Surgeon-General 1964). The 1964 ban on cigarette advertising on television was the first significant official restriction on the tobacco industry. In the early 1960s the proportion of adult male smokers declined from about 60 per cent to reach a level of around 52–53 per cent, where it stuck until the early 1970s. The proportion of women smokers remained fairly steady just above 40 per cent as did the tendency of manual workers to smoke more than professionals. The second edition of the RCP report in 1971 further implicated smoking in other forms of malignancy, respiratory and heart diseases, and complications of pregnancy (RCP 1971). It called for further restrictions on the advertising and sale of cigarettes (including warning notices on packets) and for bans on smoking in public places. This report prompted the formation of the campaigning group Action on Smoking and Health (ASH) which gave the anti-smoking cause a higher media profile. Smoking levels fell steadily from the early 1970s, among men and women, to reach a plateau at around 28 per cent in the mid-1990s. The discovery of the dangers of ‘passive smoking’ in the 1980s marked the third phase of the tobacco wars and a decisive shift in the anti-smoking campaign. The first indication of this problem came in a paper from Japan in 1981; by 1986 the US Surgeon- General noted that some thirteen studies from five different countries had confirmed an increased risk (US Surgeon-General 1986). The resulting 1987 ban on smoking on US domestic air flights and the attendant controversy put the passive smoking issue decisively on the public agenda. In 1988, the Froggat Committee, an independent scientific committee on smoking and health, estimated that passive smoking caused an increased risk of lung cancer of between 10 and 30 per cent and recommended restrictions on smoking in workplaces and in public (Jackson 1995). In 1992 the US Environmental Protection Agency declared ‘environmental tobacco smoke’ (ETS) a carcinogen, or cancer-causing agent (US EPA 1992). In 1997 the California Environmental Protection Agency added low birth weight babies, cot death, childhood asthma and nasal sinus cancer 38 THE REGULATION OF LIFESTYLE to the list of conditions caused by ETS (California EPA 1997). British meta-analyses confirmed increased risks of lung cancer (24 per cent) and coronary heart disease (23 per cent) (Hackshaw et al. A re-analysis of the same studies three years later acknowledged a ‘modest degree of publication bias’ (a result of the fact that studies which reveal no increased risk are less likely to be published) and adjusted the excess risk of lung cancer down from 24 per cent to 15 per cent (Copas, Shi 2000) The case against ETS transformed smoking from a self- endangering choice into an anti-social act. The smoker was not only engaging in a personally destructive practice, but one which was polluting the immediate environment and threatening a cast of ‘innocent victims’—non-smoking spouses (generally wives), children, unborn babies. Parental smoking came to be regarded as little better than child abuse (indeed it soon became a significant barrier to adoption). The campaign led to the establishment of ‘smoke-free’ areas and then smoking bans in workplaces, on public transport and other public spaces. The award, by Stockport council in 1993, of £15,000 in damages to Veronica Bland, who claimed that her chronic bronchitis resulted from eleven years of exposure to smoking workmates, marked the public affirmation of the status of the passive smoker in Britain. As the medical historian Allan Brandt observed, ‘in less than a decade, American public space was radically subdivided on the basis of the harms of passive smoking’ (Brandt 1998). Despite the growing medical (and political) consensus about the dangers of passive smoking, the issue has remained controversial. The Swedish toxicologist Robert Nilsson, while accepting the plausibility of the ETS-lung cancer link and the fact that numerous studies appear to show a statistically significant increase in risk, has questioned its epidemiological significance (Nilsson 1997). Thus he offered estimates, on the basis of current knowledge, of the annual incidence of cancer in a population of 100,000 resulting from various environmental factors: unknown (177), diet (135), smoking (68), other lifestyle factors (45), sunshine (23), …ETS (2). By contrast, in a population which consumes Japanese seafood (which contains arsenic) this will cause 12 cases of cancer; where there are traces of natural arsenic in drinking water, this will cause five cases; eating mushrooms will cause three cases.
Unfortunately generic levitra soft 20mg otc erectile dysfunction in the young, it has proved very difﬁcult to exploit these properties quality 20 mg levitra soft natural treatment erectile dysfunction exercise, due both to the cost and scarcity of large natural diamonds, and the fact that diamond was only available in the form of stones or grit. It had been known for 200 years that diamond is composed solely of carbon, and many attempts were made to synthesise diamond artiﬁcially using as a starting material another commonly occurring form of carbon, graphite. This proved extremely difﬁcult, mainly because at room temperature and pres- sure, graphite is more stable than diamond. Although the difference in stability between the two forms of carbon is actually quite small, their structures are so different that it would require a large amount of energy to convert between them. Ironically, this large energy barrier which makes diamond so rare is also responsible for its existence, since diamond, once Diamond thin films 77 formed, cannot spontaneously convert to the more stable graphite phase. To overcome these problems, researchers realised that in order to form diamond, they had to choose conditions where diamond, and not graphite, is the more stable phase. The knowledge of the conditions under which natural diamond is formed deep underground, suggested that diamond could be formed by heating carbon under extreme pressure. This process forms the basis of the so-called high-pressure high-temperature growth technique, ﬁrst marketed by General Electric, and which has been used to produce ‘industrial diamond’ for several decades. In this process, graphite is compressed in a hydraulic press to tens of thousands of atmospheres, heated to over 2000°C in the presence of a suitable metal catalyst, and left until diamond crystallises. The diamond crystals this produces are used for a wide range of industrial processes which utilise the hardness and wear resistance properties of diamond, such as cutting and machining mechan- ical components, and for polishing and grinding of optics. However, the drawback of this method is that it still produces diamond in the form of single crystals ranging in size from nanometres to millimetres, and this limits the range of applications for which it can be used. What is required is a method to produce diamond in a form which can allow many more of its superlative properties to be exploited – in other words, diamond in the form of a thin ﬁlm. These ideas led to experiments in which carbon-containing gases were heated under reduced pressure until the molecules broke apart, and then these fragments were condensed onto a nearby surface. Analysis showed that the thin ﬁlm that resulted from this did, indeed, contain diamond. However, the rate of growth in these early experiments was low, and the ﬁlms were impure, containing a large proportion of unwanted graphite. The breakthrough came in the late 1960s, when researchers in the USA discovered that the presence of atomic hydrogen during the deposi- tion process would remove graphite from a surface much faster than diamond. In diamond, every carbon atom is bonded to four others in a strong, rigid tetrahedral structure. This process became known as ‘chemical vapour deposition’ (CVD), since it involves a chemi- cal reaction occurring within a vapour over a surface, leading to deposition of a thin coating onto that surface. Over the next few years more break- throughs were made which allowed diamond ﬁlms to be grown at signiﬁ- cant rates on many useful materials. This series of discoveries stimulated world-wide interest in diamond CVD, in both academia and industry, which continues to the present day. While each method differs in detail, they all share a number of features in common. For example, growth of diamond (rather than graph- ite) normally requires that the precursor gas (usually methane, CH4) is diluted in excess of hydrogen – typically the mixing ratio is 1 per cent methane to 99 per cent hydrogen. Also, the temperature of the substrate is usually greater than 700°C in order to ensure the formation of diamond rather than amorphous carbon.